Cytokines and HCV-Related Disorders

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Cytokines and HCV-Related Disorders

Cytokines are intercellular mediators involved in viral control and liver damage being induced by infection with hepatitis C virus (HCV). The complex cytokine network operating during initial infection allows a coordinated, effective development of both innate and adaptive immune responses. However, HCV interferes with cytokines at various levels and escapes immune response by inducing a T-help...

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HCV-Related Nervous System Disorders

Chronic infection with hepatitis C virus (HCV) is associated with a wide spectrum of extrahepatic manifestations, affecting different organ systems. Neurological complications occur in a large number of patients and range from peripheral neuropathy to cognitive impairment. Pathogenetic mechanisms responsible for nervous system dysfunction are mainly related to the upregulation of the host immun...

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HCV-related autoimmune and neoplastic disorders: the HCV syndrome.

Hepatitis C virus (HCV) chronic infection may be associated with a great number of both hepatic and extrahepatic manifestations. HCV lymphotropism is responsible for poly-oligoclonal B-lymphocyte expansion, which is the common underlying alteration in a significant percentage of HCV-infected individuals. The consequent production of different autoantibodies and immune-complexes, including cryog...

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Immune-Related Disorders and Extrahepatic Diseases in Chronic HCV Infection

Hepatitis C virus (HCV) represents one of the most important causes of chronic active liver disease worldwide, potentially resulting in cirrhosis, and hepatocellular carcinoma. HCV is mainly characterized by two major immunologic fingerprints, namely, escape of immune response in more than 80% of infected patients and production of autoanti-bodies in almost half of them. Within the infected hos...

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HCV-Related Central and Peripheral Nervous System Demyelinating Disorders

Chronic infection with hepatitis C virus (HCV) is associated with a large spectrum of extrahepatic manifestations (EHMs), mostly immunologic/rheumatologic in nature owing to B-cell proliferation and clonal expansion. Neurological complications are thought to be immune-mediated or secondary to invasion of neural tissues by HCV, as postulated in transverse myelitis and encephalopathic forms. Prim...

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ژورنال

عنوان ژورنال: Clinical and Developmental Immunology

سال: 2012

ISSN: 1740-2522,1740-2530

DOI: 10.1155/2012/468107